Dr.Rahul Parmar resident in orthopaedics

Ortho?gynaec?? Or just mbbs!

2010-05-17T00:14:00.000+05:30

1month yet to go to ve a clear view of my next 3yrs study..i hope i ll cope up the ortho thing..here in truma centre i m having ortho residency ..much work but lets see how it goes..thank you all for support...

GUJ UNI COUNSELLING DATES

2010-03-27T18:10:00.000+05:30

BJ MEDICAL college.. counselling date 6-4-2010 .....

more pg seats..

2010-03-18T01:22:00.000+05:30

AHMEDABAD: The Medical Council of India has gifted wannabe doctors of Gujarat with 290 more seats for post-graduate medical courses. Nationwide, the seat increase is 3,791.

The change is effective from the academic year 2010-11. This jump in seats is attributed to a new set of rules introduced by MCI recently where every professor can take care of two students. Earlier, one professor was limited to one student. This has effectively doubled the capacity.

Correspondingly, the number of PG seats has jumped to 18,000. The primary beneficiaries of this seat increase will be government-run medical colleges like BJ Medical College and NHL Municipal Medical College in Ahmedabad, Vadodara Medical College and government medical colleges in Surat, Rajkot, Bhavnagar and Jamnagar.

Nationwide, Andhra Pradesh has bagged the most seats with 611, followed by Maharashtra (476). Gujarat is at No.6 with 290 seats.

"Students hailing from poor economic backgrounds will benefit from this increase. Now, deserving students can get admission without having to worry about exorbitant capitation fees. The students will no longer need to contend with hefty fee structure of private colleges," said Ketan Desai, president of MCI.

reikii

2010-03-07T15:35:00.003+05:30

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Today is

2009-09-21T10:18:00.000+05:30

World alzheimer's day...and this year's theme is DIAGNOSE ALZHEIMER:SEE IT SOONER....

FREE mock tests THRISSUR P.G ADMISSION

2009-07-25T22:49:00.002+05:30

http://www.medpgthrissur.com/online/login/login.php

THIS WEBSITE IS TOTALY FREE AND IT CONDUCTS EXAMS EVERY TUE/SAT FROM 8 TO 9 PM ......

free mock tests for medicos

2009-07-24T21:21:00.002+05:30

just get urself to prepgmedicos.com

they r shceduling all the subject wise tests too...

cerebellum cell types

2009-06-02T01:45:00.002+05:30

Cerebellum: Cell Types

Name	Target (Axon Termination)	Transmitter	Function
Purkinje cell	Deep cerebellar nuclei	GABA	Inhibitory*
Granule cell	Purkinje cell	Glutamate	Excitatory
Stellate cell	Purkinje cell	GABA	Inhibitory
Basket cell	Purkinje cell	GABA	Inhibitory
Golgi cell	Granule cell	GABA	Inhibitory

*Purkinje cells are the only outflow from the cerebellar cortex.

Facial nerve branches(out side skull)

2009-04-21T20:07:00.003+05:30

Two Zulus Buggered My Cat.

Five major facial branches (in parotid gland) - from top to bottom:

day of appearing rash after fever

2009-03-04T00:16:00.005+05:30

very varicella( rubella) 1st day

sick scarlet fever 2nd day

person small Pox 3rd day

must measles 4th day

take typhus 5th day

double dengue 6th day

treatment typhoid 7 th day

my notes--platelet guidelines

2009-02-28T19:50:00.004+05:30

Platelet Transfusion Guidelines

Name: Platelets

Major Products Available:

Platelets (Random donor platelets - RDP)

Platelets Pooled

Platelets Pheresis (Single donor platelets - SDP)

These products can be leukoreduced, washed, and/or irradiated.

Description/Contents: RDP are separated from whole blood by differential centrifugation. SDP are harvested from a single donor by hemapheresis. One unit of RDP contains at least 5.5x10¹⁰ platelets, typically 7.5x10¹⁰ platelets. Pooled RDP are typically prepared from 4-6 units of RDP. One unit of SDP contains at least 3x10¹¹platelets, typically 4x10¹¹platelets. Platelets are suspended in donor plasma, unless washed. Volume is specified on the label.

Indications:

Prevention/treatment of non-surgical bleeding due to thrombocytopenia.

If possible, prior to transfusion the reason for thrombocytopenia should be established. When thrombocytopenia is caused by marrow failure, the following transfusion triggers are considered appropriate:

If platelet count is <10,000/mL and no additional abnormalities exist.

If platelet count is between 10,000 and 20,000/mL and coagulation abnormalities exist or there are extensive petechiae or ecchymoses.
If the patient is bleeding at sites other than skin and platelet count is <40-50,000/m L.

Patients with accelerated platelet destruction with significant bleeding (such as autoimmune thrombocytopenia or drug-induced thrombocytopenia).

The endpoint should be cessation of bleeding, since an increment in platelet count is not likely to be achieved. Prophylactic transfusion is not indicated in these disorders.

Prior to surgical and major invasive procedures when the platelet count is <50,000/ mL.

During neurosurgical and ophthamologic procedures some authorities recommend that the platelet count be maintained between 70,000 and 100,000/mL.

Bleeding with qualitative platelet defect documented by history and/or laboratory tests.

The cause should be identified and corrected, if possible, prior to surgery. Platelet transfusion is indicated only if the defect cannot be otherwise corrected: for example, a congenital platelet abnormality. Consultation with the blood bank physician is recommended in these situations.

Diffuse microvascular bleeding after cardiopulmonary bypass or massive transfusion.

Platelet count and coagulation studies should be performed prior to the transfusion to guide subsequent therapy. During surgery on patients with quantitative or qualitative platelet defects, the adequacy of hemostasis in patients should be evaluated by the assessment of microvascular bleeding.

Platelet transfusion should be avoided in thrombotic thrombocytopenic purpura, heparin-induced thrombocytopenia, and post-transfusion purpura, except in cases of life-threatening hemorrhage.

Dosage and administration: Compatibility testing is not required. Platelet concentrate products should be ABO identical where possible because platelet increments may be higher. If not possible, good clinical results are usually obtained with ABO mismatched platelets. In this case, transfusion of large quantities of ABO incompatible plasma may lead to a positive direct antiglobulin test and, rarely, clinically significant red cell destruction. Rh compatibility is important but not always possible. Post-exposure prophylaxis with anti-Rh immune globulin should be considered following Rh positive platelet product transfusions to Rh negative women who may have children in the future.

A typical platelet transfusion dose is 1 SDP or 4-5 pooled RDP. This should raise the platelet count of a typical 70 kg man approximately 30,000-50,000/mL. Platelet count increments after transfusion may be lower than expected in the presence of certain medications, fever, splenomegaly, infection, or alloimmunization to HLA or specific platelet antigens. Consult the Blood Bank/Transfusion Medicine physician or Hematologist in such cases.

Alternative Therapy: DDAVP (desmopressin) may improve the platelet functional defect in uremia. It also raises von Willebrand factor levels in mild-moderate von Willebrand's disease, which may improve platelet function. Pharmacologic agents such as aprotinin may reduce major surgical bleeding and thereby avoid the dilutional thrombocytopenia characteristic of massive transfusion. Some of these agents may also have a direct effect on improving platelet function.

14 feb a new beginning!!

2009-02-13T23:37:00.001+05:30

abt to start a new job.... and looking forward for first challange of my life...